Return-Path: <nifl-ld@literacy.nifl.gov> Received: from literacy (localhost [127.0.0.1]) by literacy.nifl.gov (8.10.2/8.10.2) with SMTP id j2755QC00931; Mon, 7 Mar 2005 00:05:26 -0500 (EST) Date: Mon, 7 Mar 2005 00:05:26 -0500 (EST) Message-Id: <000901c522d2$f9537540$d60a1845@DeepThought> Errors-To: listowner@literacy.nifl.gov Reply-To: nifl-ld@literacy.nifl.gov Originator: nifl-ld@literacy.nifl.gov Sender: nifl-ld@literacy.nifl.gov Precedence: bulk From: "Woods" <woods@ncia.net> To: Multiple recipients of list <nifl-ld@literacy.nifl.gov> Subject: [NIFL-LD:4593] Re: Dyslexia Research X-Listprocessor-Version: 6.0c -- ListProcessor by Anastasios Kotsikonas X-Mailer: Microsoft Outlook Express 6.00.2900.2180 Content-Transfer-Encoding: 7bit Content-Type: text/plain; Status: O Content-Length: 697 Lines: 15 I can see where one day we might know more about how the genes express themselves. Knowing that would be infinitely more useful than just knowing the name of a gene involved in dyslexia or some other condition. Such knowledge might give us insight on targeting specific kinds of remediation and not waste time on ineffective approaches. For instance, if Mary has the 'sees things upside down' gene, we might then know to not to give her books right side up, and we wouldn't make her spend her life working on word attack and using color overlays. Tom Woods > The writer asks an interesting question. What implications could this > have > for our work with adults that have dyslexia?
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